







Software in the natural world: A computational approach to hierarchical emergence
Understanding the functional architecture of complex systems is crucial to illuminate their inner workings and enable effective methods for their prediction and control. Recent advances have introduced tools to characterise emergent macroscopic levels; however, while these approaches are successful in identifying when emergence takes place, they are limited in the extent they can determine how it does. Here we address this limitation by developing a computational approach to emergence, which characterises macroscopic processes in terms of their computational capabilities. Concretely, we articulate a view on emergence based on how software works, which is rooted on a mathematical formalism that articulates how macroscopic processes can express self-contained informational, interventional, and computational properties. This framework establishes a hierarchy of nested self-contained processes that determines what computations take place at what level, which in turn delineates the functional architecture of a complex system. This approach is illustrated on paradigmatic models from the statistical physics and computational neuroscience literature, which are shown to exhibit macroscopic processes that are akin to software in human-engineered systems. Overall, this framework enables a deeper understanding of the multi-level structure of complex systems, revealing specific ways in which they can be efficiently simulated, predicted, and controlled.

Dynamical independence: Discovering emergent macroscopic processes in complex dynamical systems
We introduce a notion of emergence for macroscopic variables associated with highly multivariate microscopic dynamical processes. Dynamical independence instantiates the intuition of an emergent macroscopic process as one possessing the characteristics of a dynamical system ``in its own right,'' with its own dynamical laws distinct from those of the underlying microscopic dynamics. We quantify (departure from) dynamical independence by a transformation-invariant Shannon information-based measure of dynamical dependence. We emphasize the data-driven discovery of dynamically independent macroscopic variables, and introduce the idea of a multiscale ``emergence portrait'' for complex systems. We show how dynamical dependence may be computed explicitly for linear systems in both time and frequency domains, facilitating discovery of emergent phenomena across spatiotemporal scales, and outline application of the linear operationalization to inference of emergence portraits for neural systems from neurophysiological time-series data. We discuss dynamical independence for discrete- and continuous-time deterministic dynamics, with potential application to Hamiltonian mechanics and classical complex systems such as flocking and cellular automata.
Unified framework for information integration based on information geometry
Significance Measuring the degree of causal influences among multiple elements of a system is a fundamental problem in physics and biology. We propose a unified framework for quantifying any combination of causal relationships between elements in a hierarchical manner based on information geometry. Our measure of integration, called geometrical integrated information, quantifies the strength of multiple causal influences among elements by projecting the probability distribution of a system onto a constrained manifold. This measure overcomes mathematical problems of existing measures and enables an intuitive understanding of the relationships between integrated information and other measures of causal influence such as transfer entropy. Inspired by the integration of neural activity in consciousness studies, our measure should have general utility in analyzing complex systems. , Assessment of causal influences is a ubiquitous and important subject across diverse research fields. Drawn from consciousness studies, integrated information is a measure that defines integration as the degree of causal influences among elements. Whereas pairwise causal influences between elements can be quantified with existing methods, quantifying multiple influences among many elements poses two major mathematical difficulties. First, overestimation occurs due to interdependence among influences if each influence is separately quantified in a part-based manner and then simply summed over. Second, it is difficult to isolate causal influences while avoiding noncausal confounding influences. To resolve these difficulties, we propose a theoretical framework based on information geometry for the quantification of multiple causal influences with a holistic approach. We derive a measure of integrated information, which is geometrically interpreted as the divergence between the actual probability distribution of a system and an approximated probability distribution where causal influences among elements are statistically disconnected. This framework provides intuitive geometric interpretations harmonizing various information theoretic measures in a unified manner, including mutual information, transfer entropy, stochastic interaction, and integrated information, each of which is characterized by how causal influences are disconnected. In addition to the mathematical assessment of consciousness, our framework should help to analyze causal relationships in complex systems in a complete and hierarchical manner.

Causality
Written by one of the preeminent researchers in the field, this book provides a comprehensive exposition of modern analysis of causation. It shows how causality has grown from a nebulous concept into a mathematical theory with significant applications in the fields of statistics, artificial intelligence, economics, philosophy, cognitive science, and the health and social sciences. Judea Pearl presents and unifies the probabilistic, manipulative, counterfactual, and structural approaches to causation and devises simple mathematical tools for studying the relationships between causal connections and statistical associations. Cited in more than 2,100 scientific publications, it continues to liberate scientists from the traditional molds of statistical thinking. In this revised edition, Judea Pearl elucidates thorny issues, answers readers' questions, and offers a panoramic view of recent advances in this field of research. Causality will be of interest to students and professionals in a wide variety of fields. Dr Judea Pearl has received the 2011 Rumelhart Prize for his leading research in Artificial Intelligence (AI) and systems from The Cognitive Science Society.

The Dimensions of Emergent Spacetime in the Influence Network
The Dimensions of Emergent Spacetime in the Influence Network KEVIN KNUTH, State Univ of NY -Albany -It has been previously demonstrated that the consistent quantification of a causally ordered set of events (influence network) with respect to
The Abstraction Fallacy: Why AI Can Simulate But Not Instantiate Consciousness
Computational functionalism dominates current debates on AI consciousness. This is the hypothesis that subjective experience emerges entirely from abstract causal topology, regardless of the underlying physical substrate. We argue this view fundamentally mischaracterizes how physics relates to information. We call this mistake the Abstraction Fallacy. Tracing the causal origins of abstraction reveals that symbolic computation is not an intrinsic physical process. Instead, it is a mapmaker-dependent description. It requires an active, experiencing cognitive agent to alphabetize continuous physics into a finite set of meaningful states. Consequently, we do not need a complete, finalized theory of consciousness to assess AI sentience—a demand that simply pushes the question beyond near-term resolution and deepens the AI welfare trap. What we actually need is a rigorous ontology of computation. The framework proposed here explicitly separates simulation (behavioral mimicry driven by vehicle causality) from instantiation (intrinsic physical constitution driven by content causality). Establishing this ontological boundary shows why algorithmic symbol manipulation is structurally incapable of instantiating experience. Crucially, this argument does not rely on biological exclusivity. If an artificial system were ever conscious, it would be because of its specific physical constitution, never its syntactic architecture. Ultimately, this framework offers a physically grounded refutation of computational functionalism to resolve the current uncertainty surrounding AI consciousness.
Cognition in a Social Context: A Social-Interactionist Approach to Emergent Phenomena
The formation of collective memories, emotions, and beliefs is a fundamental characteristic of human communities. These emergent outcomes are thought to be the result of a dynamical system of communicative interactions among individuals. But despite recent psychological research on collective phenomena, no programmatic framework to explore the processes involved in their formation exists. Here, we propose a social-interactionist approach that bridges cognitive and social psychology to illuminate how microlevel cognitive phenomena give rise to large-scale social outcomes. It involves first establishing the boundary conditions of cognitive phenomena, then investigating how cognition is influenced by the social context in which it is manifested, and finally studying how dyadic-level influences propagate in social networks. This approach has the potential to (a) illuminate the large-scale consequences of well-established cognitive phenomena, (b) lead to interdisciplinary dialogues between psychology and the other social sciences, and (c) be more relevant for public policy than existing approaches.

Local Causation
The counterfactual and regularity theories are universal accounts of causation. I argue that these should be generalized to produce local accounts of causation. A hallmark of universal accounts of causation is the assumption that apparent variation in causation between locations must be explained by differences in background causal conditions, by features of the causal-nexus or causing-complex. The local account of causation presented here rejects this assumption, allowing for genuine variation in causation to be explained by differences in location. I argue that local accounts of causation are plausible, and have pragmatic, empirical and theoretical advantages over universal accounts. I then report on the use of presheaves as models of local causation. The use of presheaves as models of local variation has precedents in algebraic geometry, category theory and physics; they are here used as models of local causal variation. The paper presents this idea as stemming from an approach using presheaves as models of local truth. Finally, I argue that a proper balance between universal and local causation can be assuaged by moving from presheaves to fully-fledged sheaf models.
Are biological systems poised at criticality?
Many of life's most fascinating phenomena emerge from interactions among many elements--many amino acids determine the structure of a single protein, many genes determine the fate of a cell, many neurons are involved in shaping our thoughts and memories. Physicists have long hoped that these collective behaviors could be described using the ideas and methods of statistical mechanics. In the past few years, new, larger scale experiments have made it possible to construct statistical mechanics models of biological systems directly from real data. We review the surprising successes of this "inverse" approach, using examples form families of proteins, networks of neurons, and flocks of birds. Remarkably, in all these cases the models that emerge from the data are poised at a very special point in their parameter space--a critical point. This suggests there may be some deeper theoretical principle behind the behavior of these diverse systems.

Gene regulatory networks: from correlative models to causal explanations
Gene regulatory networks (GRNs) explain how the genome controls cellular behaviour and tissue morphogenesis, serving to connect molecular mechanism to functional output. Single-cell technologies now provide descriptions of these networks with unprecedented detail, but this advance has also revealed gene regulatory systems that are too complex for our existing conceptual frameworks. GRNs, which should provide mechanistic explanations, are increasingly reduced to statistical correlations — ‘hairballs’ that fail to capture molecular causation. Here, we explore why this dilemma exists and propose a path forward. We argue that methods in ‘representation learning’ can be used to model GRNs, without needing to capture every molecular detail. For this framework, we advocate three linked principles: models must be inherently mechanistic, with structures grounded in cellular and evolutionary biology; molecular principles and constraints must be used to reduce the solution space for learning GRN models; and more sophisticated forms of experimental perturbation and synthetic biological engineering are needed to train models and test predictions. By reimagining GRNs through these principles, we can bridge the gap from data abundance to new conceptual understanding.

Alexander Lerchner, The Abstraction Fallacy: Why AI Can Simulate But Not Instantiate Consciousness - PhilPapers
Computational functionalism dominates current debates on AI consciousness. This is the hypothesis that subjective experience emerges entirely from abstract causal topology, regardless of the underlying physical substrate. We argue this view ...

AI Data Centers Will Be Obsolete (Geometric Reasoning Explained)
Alexander Lerchner, The Abstraction Fallacy: Why AI Can Simulate But Not Instantiate Consciousness - PhilArchive
Computational functionalism dominates current debates on AI consciousness. This is the hypothesis that subjective experience emerges entirely from abstract causal topology, regardless of the underlying physical substrate. We argue this view ...

Systems of Global Governance in the Era of Human-Machine Convergence
Technology is increasingly shaping our social structures and is becoming a driving force in altering human biology. Besides, human activities already proved to have a significant impact on the Earth system which in turn generates complex feedback loops between social and ecological systems. Furthermore, since our species evolved relatively fast from small groups of hunter-gatherers to large and technology-intensive urban agglomerations, it is not a surprise that the major institutions of human society are no longer fit to cope with the present complexity. In this note we draw foundational parallelisms between neurophysiological systems and ICT-enabled social systems, discussing how frameworks rooted in biology and physics could provide heuristic value in the design of evolutionary systems relevant to politics and economics. In this regard we highlight how the governance of emerging technology (i.e. nanotechnology, biotechnology, information technology, and cognitive science), and the one of climate change both presently confront us with a number of connected challenges. In particular: historically high level of inequality; the co-existence of growing multipolar cultural systems in an unprecedentedly connected world; the unlikely reaching of the institutional agreements required to deviate abnormal trajectories of development. We argue that wise general solutions to such interrelated issues should embed the deep understanding of how to elicit mutual incentives in the socio-economic subsystems of Earth system in order to jointly concur to a global utility function (e.g. avoiding the reach of planetary boundaries and widespread social unrest). We leave some open questions on how techno-social systems can effectively learn and adapt with respect to our understanding of geopolitical complexity.

Model Collapse Ends AI Hype
#precision #scaling #generalization #impact | Jean-Rémi King
Well, we did not expect that: in 10 days: 1.6K ⭐ on Github, 70K downloads on HuggingFace 🤗, >6M views on socials. 🧠 TRIBE v2, our new foundation model of the human brain responses to sight, sound, and language, has led to a surge of community demos, dozens of PRs and issues, and a level of engagement that rivals major flagships model at Meta (e.g. Llama4: 3.6M views, and DINOv3: 900K): https://lnkd.in/eQgqyDvf As this raised several questions, let me emphasize some elements of clarification: 1. #Precision: This model exclusively uses fMRI recordings. While powerful, these data are slow-paced proxy measurements of brain activity, and aggregate responses ~100k-3M neurons per data point. This means we are very far from a neuronal-level modeling of the brain. 2. #Scaling: We leveraged ~1,000 hours of naturalistic fMRI data. This is very substantial for a functional imaging study with naturalistic data; but still this is "small data" compared to medical or biology foundation models in general (e.g. structural MRI models are typically tens of thousands of individual brains). 3. #Generalization: The model shows surprising out-of-domain generalization, but it is **not** a replacement for new data collection. The model will almost surely fail in areas it hasn't seen: task-specific behaviors, memory protocols, touch sensation etc. And these are the "known unknowns" - I expect we'll discover many more unknown unknowns along the way. 4. #Impact: The goal of this model, and of our team in general, is fundamental research in neuroscience. While I am optimistic about how such foundation models will help clinical diagnosis, prognosis, and patient care, the physics of MRI is such that there is no clear path for making this kind of technology directly usable to consumer/wearable products. We're in for the science, hence the open sourcing: 📄 Paper: https://lnkd.in/e7cbunJp 💻 Code: https://lnkd.in/ebwBVuJp ▶️ Demo: https://lnkd.in/eEUVxP4S 🤗 Model: https://lnkd.in/e2T8nPJP
Causal interactions within complex systems can be analyzed at multiple spatial and temporal scales. For example, the brain can be analyzed at the l...